• This product is strictly for research purposes only. Not for human use.
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TB-500

Price range: $80.00 through $120.00

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TB-500 is studied for its role in actin regulation and tissue repair processes. Derived from thymosin beta-4, it may promote cell migration, reduce inflammation, and support muscle and connective tissue recovery. Its regenerative properties make it a key peptide in recovery-focused research.

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Overview

Research suggests that TB-500 may support tissue repair and cellular regeneration by influencing actin regulation and promoting wound healing pathways. Derived from the naturally occurring protein thymosin beta-4, it has gained attention for its potential role in enhancing muscle recovery, reducing inflammation, and supporting tendon and ligament repair. Studies indicate it may help modulate cell migration and tissue remodeling processes. Researchers continue to explore its applications in recovery, regenerative medicine, and musculoskeletal support. For an in-depth look, see our TB-500 research guide.

Key Characteristics

Molecular Details

  • Chemical Name: TB-500 (Synthetic Thymosin Beta-4 fragment)

  • Parent Molecule: Thymosin Beta-4

  • Molecular Formula (Thymosin Beta-4): C₂₁₂H₃₅₀N₅₆O₇₈S

  • Molecular Weight: ~4,963 g/mol (full peptide)

  • Structure: Synthetic peptide fragment derived from the naturally occurring 43-amino acid Thymosin Beta-4 protein

Product Information

  • Form: Lyophilized peptide powder

  • Solubility: Soluble in sterile water or bacteriostatic water prior to use

  • Stability: Stable under refrigeration; limited stability once reconstituted

  • Half-Life: Variable depending on formulation and route; biologic activity extends beyond plasma presence

  • Storage: Store refrigerated at 36–46°F (2–8°C); protect from light and moisture

How It Works

TB-500 (no DAC) operates by binding to growth hormone secretagogue receptors in the pituitary gland, much like a specialized messenger delivering instructions to a command center. ok

Primary Mechanisms

Cell Migration Regulation

Promotes actin binding and cytoskeletal remodeling to support cellular movement

Angiogenesis Stimulation

Encourages new blood vessel formation in damaged tissue models

Tissue Remodeling Support

Enhances extracellular matrix organization during repair phases

Inflammatory Modulation

Supports balanced cytokine signaling during recovery processes

Research Benefits

Muscle & Connective Tissue Recovery

Demonstrates accelerated repair in muscle, tendon, and ligament models

Improved Vascularization

Increases capillary density and tissue perfusion in injury studies

Wound Healing Enhancement

Supports faster dermal closure and organized collagen deposition

Research Findings

Safety considerations and tolerability profile observed in research settings.

Musculoskeletal Repair

Research indicates:

  • Increased capillary density in injured tissue

  • Enhanced endothelial cell migration

  • Improved tissue perfusion during healing phases

Wound Healing

Evidence shows:

  • Faster dermal wound closure

  • Enhanced collagen deposition and remodeling

  • Reduced scar tissue formation markers

Angiogenesis & Vascular Effects

Research indicates:

  • Increased capillary density in injured tissue

  • Enhanced endothelial cell migration

  • Improved tissue perfusion during healing phases

Cardiac & Organ Tissue Studies

Preclinical research suggests:

  • Support for cardiac tissue repair following injury

  • Improved functional recovery in myocardial models

  • Reduced inflammatory damage in organ tissues

Potential Side Effects in Research

Safety considerations and tolerability profile observed in research settings.

Generally Well Tolerated

Glutathione is a naturally occurring endogenous molecule with high safety in research and supplementation contexts.

Common Observations

Temporary fatigue or headach

Monitoring Considerations

Inflammatory markers

Most safety data derive from clinical use in approved indications or controlled studies.

References

Goldstein, Allan L., et al. “Thymosin Beta-4: Actin-Sequestering Protein with Role in Wound Healing and Angiogenesis.” Annals of the New York Academy of Sciences, vol. 1112, 2007, pp. 146–153. PubMed
Smart, N., et al. “Thymosin Beta-4 Induces Adult Epicardial Progenitor Mobilization and Neovascularization.” Nature, vol. 445, 2007, pp. 177–182. PubMed
Malinda, K. M., et al. “Thymosin Beta-4 Accelerates Wound Healing.” Journal of Investigative Dermatology, vol. 113, no. 3, 1999, pp. 364–368. PubMed

THIRD-PARTY LAB TESTED

Certificate of Analysis

Independent laboratory testing confirms purity and composition

99.312%

Purity

58ABF

Batch

January 2026

Date

View Certificate →

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